Select Publications
Journal articles
2012, 'Phenotypic discrepancies in acetyl-CoA carboxylase 2-deficient mice', The Journal of Biological Chemistry, 287, pp. 15801, http://dx.doi.org/10.1074/jbc.O112.356915
,2011, 'Continued clearance of apoptotic cells critically depends on the phagocyte Ucp2 protein', Nature, 477, pp. 220 - 224, http://dx.doi.org/10.1038/nature10340
,2010, 'Mitochondrial regulation of insulin action', International Journal of Biochemistry and Cell Biology, 42, pp. 1936 - 1939, http://dx.doi.org/10.1016/j.biocel.2010.08.020
,2010, 'Genetic ablation of cyclophilin D, a component of the mitochondrial permeability transition pore, improves insulin sensitivity in high-fat fed mice', FASEB JOURNAL, 24, https://www.webofscience.com/api/gateway?GWVersion=2&SrcApp=PARTNER_APP&SrcAuth=LinksAMR&KeyUT=WOS:000208675507150&DestLinkType=FullRecord&DestApp=ALL_WOS&UsrCustomerID=891bb5ab6ba270e68a29b250adbe88d1
,2010, 'The etiology of obesity-induced insulin resistance', , pp. 205 - 238
,2010, 'Acute or chronic upregulation of mitochondrial fatty acid oxidation has no net effect on whole-body energy expenditure or adiposity', Cell Metabolism, 11, pp. 70 - 76, http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&dopt=Citation&list_uids=20074529
,2010, 'Global phosphoproteomics identifies a major role for AKT and 14-3-3 in regulating EDC3', Molecular and Cellular Proteomics, 9, pp. 682 - 694, http://dx.doi.org/10.1074/mcp.M900435-MCP200
,2010, 'Sialic acid modification of adiponectin is not required for multimerization or secretion but determines half-life in circulation', Molecular Endocrinology, 24, pp. 229 - 239, http://dx.doi.org/10.1210/me.2009-0133
,2009, 'The etiology of obesity-induced insulin resistance', , pp. 99 - 132
,2009, 'Insulin resistance is a cellular antioxidant defense mechanism', Proceedings of the National Academy of Sciences of the United States of America, 106, pp. 17787 - 17792, http://www.pnas.org/content/106/42/17787.long
,2009, 'Pigment Epithelium-Derived Factor Contributes to Insulin Resistance in Obesity', Cell Metabolism, 10, pp. 40 - 47
,2008, 'CaMKII-Mediated Phosphorylation of the Myosin Motor Myolc Is Required for Insulin-Stimulated GLUT4 Translocation in Adipocytes', Cell Metabolism, 8, pp. 384 - 398
,2008, 'IRS1-independent defects define major nodes of insulin resistance', Cell Metabolism, 7, pp. 421 - 433, http://dx.doi.org/10.1016/j.cmet.2008.04.005
,2007, 'Lipid Mediators of Insulin Resistance', Nutrition Reviews, 65, http://dx.doi.org/10.1111/j.1753-4887.2007.tb00327.x
,2007, 'Lipid mediators of insulin resistance', NUTRITION REVIEWS, 65, pp. S39 - S46, http://dx.doi.org/10.1301/nr.2007.jun.S39-S46
,2007, 'Lipid mediators of insulin resistance.', Nutrition reviews, 65
,2007, 'Inhibition of Ceramide Synthesis Ameliorates Glucocorticoid-, Saturated-Fat-, and Obesity-Induced Insulin Resistance', Cell Metabolism, 5, pp. 167 - 179, http://dx.doi.org/10.1016/j.cmet.2007.01.002
,2007, 'Excess lipid availability increases mitochondrial fatty acid oxidative capacity in muscle - Evidence against a role for reduced fatty acid oxidation in lipid-induced insulin resistance in rodents', Diabetes, 56, pp. 2085 - 2092
,2006, 'The hunger switch', The FASEB Journal, 20, http://dx.doi.org/10.1096/fasebj.20.4.a177-a
,2004, 'Regulation of insulin action by ceramide: Dual mechanisms linking ceramide accumulation to the inhibition of Akt/protein kinase B', Journal of Biological Chemistry, 279, pp. 36608 - 36615, http://dx.doi.org/10.1074/jbc.M406499200
,2004, 'Seasonal, tissue-specific regulation of Akt/protein kinase B and glycogen synthase in hibernators', AMERICAN JOURNAL OF PHYSIOLOGY-REGULATORY INTEGRATIVE AND COMPARATIVE PHYSIOLOGY, 286, pp. R498 - R504, http://dx.doi.org/10.1152/ajpregu.00509.2003
,2004, 'Seasonal, tissue-specific regulation of Akt/protein kinase B and glycogen synthase in hibernators', American Journal of Physiology - Regulatory Integrative and Comparative Physiology, 286, http://dx.doi.org/10.1152/ajpregu.00509.2003
,2003, 'Assaying AKT/protein kinase B activity.', Methods in molecular medicine, 83, pp. 137 - 144, http://dx.doi.org/10.1385/1-59259-377-1:137
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